In this study, we examined the relationship between symptoms, indices of substance abuse, prior history of violence and criminally violent victimisation in a cohort of male patients with schizophrenia spectrum disorders who were resident in the community in six Victorian public mental health services. We did not include females in the sample as we there are gender differences in the nature of victimisation
[
11] and we were interested in violent victimisation which is seen more frequently in men
[
11]. In this cohort 25% of the sample had been a victim of a violent offence. The latter figure is similar to data from the UK where 23% were victims of violent crime
[
15]. A systematic review of the literature suggested criminal victimisation rates (based on self report) range from 4.3%–35% depending on the time period understudy
[
38].
In our comparison of those who were victims of violent crime and those who were not based on the crime database, we found no significant group differences in the demographic characteristics of the samples. Previous studies have found an association between unemployment and victimisation
[
13,
23,
24]. Given that the unemployment was approaching significance it is likely this factor would have reached statistical significance with a larger sample size. Studies looking at the relationship between age and violent victimisation have produced mixed findings. While there are reports that young age is a risk factor for victimisation [e.g.
[
15,
21,
22,
24,
27], we did not find this to be the case and our findings fit with other reports that young age may not be robustly associated with victimisation [e.g.
[
14,
26]. Differences between the samples under study and the lifetime measure of victimisation may, however, account for the divergent findings.
Unlike previous studies [e.g.
[
15,
21,
26,
27], we did not find a robustly significant relationship between early illness onset and violent victimisation. Honkonen et al.
[
22] reported that a shorter duration of illness was associated with increased risk for violent victimisation within a three year period. Differences in the timeframes in which violent victimisation is examined may account for the divergent findings.
In this sample we did not find any significant relationship between symptoms and violent victimisation status. It has been suggested that the association between positive symptoms and criminally violent victimisation is more apparent in the acute phase of illness when patients may present as either vulnerable or behaviourally disturbed during their community interactions
[
26]. The latter notion is consistent with some
[
14,
22], but not all previous studies
[
15,
21,
23,
30]. It is likely that the divergent findings in the literature may reflect differences in the nature of the samples studied, methodology, and the timeframe in which violent victimisation was examined. In this study we looked at lifetime victimisation in a cohort of clinically stable patients making it difficult to draw definitive conclusions on the role of the acute phase of illness in their histories of victimisation.
In line with our
a priori hypothesis, we found significant associations between substance abuse and victimisation, with significantly higher DAST scores in the violently victimized group. In particular, we found that lifetime poly-substance abuse was significantly more prevalent in those who were victims of violent crime, as was an arrest for a drug related offence. In this study, the mean age of onset for drug or alcohol use did not differ significantly between those who were and those who were not victims of violent crime. We would have thought that the latter may be a significant factor as the early onset of substance abuse tends to be associated with antisocial personality pathology which is in turn is known to be a risk factor for both violence and violent victimisation
[
15,
21]. As this is the first study to investigate a relationship between age of substance use onset and violent victimisation, this is an area that warrants further study before any definitive conclusions can be drawn on the significance of early onset substance in the victimisation literature. Overall our findings largely fit with previous reports that substance abuse problems are a risk factor for victimisation in those with major mental disorders such as schizophrenia
[
9,
12,
14,
15,
22,
23,
27,
30]. Although previous studies have found an association between alcohol abuse and victimisation [e.g.
[
9,
22,
23,
27], we did not find that this was the case in this sample. It is possible that the high rates of alcohol abuse problems in both groups may have masked any significant findings. It may also reflect differences in alcohol use measurement between studies. Alcohol abuse has been recognised as a significant correlate of violent victimisation in a recent systematic review
[
38] and we would have anticipated that it would be a significant correlate of violent victimisation. It is possible that a more in-depth measure of the extent of alcohol abuse may have resulted in more notable difference between groups. Further work is needed to clarify the specificity of different types of substance abuse problems in the violent victimisation literature.
In this study, we found no association between violent crime victimisation and perpetration. This is not consistent with previous studies documenting an association between violence and violent victimisation in patients with major mental disorder
[
10,
12,
13,
17,
19,
22-
25,
29]. Further work is therefore needed to explore potential differences in the proximal and distal factors that mediate the relationships between violence and violent victimisation in general populations and clinical samples if we are to gain a better understanding of how victim-perpetrator conflicts and interactions emerge as predominately one or the other.
Strengths and limitations
This is the first study looking at the correlates of victimisation in terms of demography, substance abuse, illness related variables and previous violence in a community sample of Australian patients with major mental illness; however, we acknowledge that there are some strengths and limitations that need to be outlined.
We used official databases as the means of assessing both violence and violent victimisation. This may have resulted in an underestimation of true violence and victimisation rates as these incidents may not be reported to police. However, there are also problems in relying on self-report due to recall bias so future work should use a combination of sources.
The study sample size was relatively small and confined to males. The findings can therefore not be extrapolated to females or mixed gender samples where gender differences in types of victimization may be apparent. This was a cross sectional study that collected data on lifetime rather than prospective post interview victimisation which makes it impossible to draw any firm conclusions on causal relationships but may also account for the lack of a significant relationship between victimisation and current symptoms. It is possible that the inclusion of patients with a greater variance in symptom scores may have resulted in the emergence of significant differences between those who were and were not victimised, but ethics approvals required the inclusion of those able to give valid consent. Prospective follow studies are needed to explore the context in which victimisation takes place in order to gain a more in-depth understanding of the nature of the victim-offender relationship and the role of active symptoms. This study adds to the evidence base that symptoms of mental illness and co-morbid substance abuse, as well as individual differences in violence potential may be important factors in victimisation, but it is important to note that there have been no notable developments in the provision of comprehensive services that also minimise risk of victimisation.