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Logo of capmcAbout manuscripts / A propos des manuscritsSubmit manuscript / soumettre un manuscrit
Biochim Biophys Acta. Author manuscript; available in PMC 2012 November 16.
Published in final edited form as:
Biochim Biophys Acta. 2008 August; 1779(8): 438–452.
Published online 2008 January 17. doi: 10.1016/j.bbagrm.2008.01.003

Table 2

An alphabetical list of pharmacological agents/treatments referred to in this paper

Chemical/treatmentEffect on Ca++ signalling
AcetylcholineAgonist of acetylcholine receptor, increase Ca++ influx
AP5Antagonist of NMDA receptor, reduce Ca++ influx
BAPTA-AMA cell permeable Ca++ chelator, reduce cytosolic Ca++
CdCl2A voltage-sensitive Ca++ channel blocker
FK506A calcineurin inhibitor
GlutamateAgonist of glutamate receptor, increase Ca++ influx through ionotropic glutamate receptors
IonomycinIonophore, increase intracellular Ca++
Ischemiaincrease intracellular Ca++
KainateIonotropic glutamate receptor agonist, increase Ca++ influx
KCl (25–50 mM)Induce Ca++ influx through Ca++ channels
KN62CaM kinase inhibitor
KN93CaM kinase inhibitor
MK-801Antagonist of NMDA receptor, reduce Ca++ influx
NifedipineL-type calcium channel blocker
NimodipineL-type calcium channel blocker
NMDAAgonist of NMDA receptor, increase Ca++ influx
PilocarpineAgonist of muscarinic acetylcholine receptor, induce seizure, increase Ca++
ThapsigarginInhibitor of the sarco/endoplasmic reticulum Ca++ ATPase, elevate cytosolic Ca++
VerapamilL-type calcium channel blocker