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Logo of genbioBioMed CentralBiomed Central Web Sitesearchsubmit a manuscriptregisterthis articleGenome BiologyJournal Front Page
 
Genome Biol. 2011; 12(11): 412.
Published online 2012 January 15. doi:  10.1186/gb-2011-12-11-412
PMCID: PMC3334595

Correction: Cell death upon epigenetic genome methylation: a novel function of methyl-specific deoxyribonucleases

Correction

We have noticed an error in the sixth paragraph of the Discussion of our article [1]. The corrected paragraph should read:

Another type IV nuclease, GmrSD, found in an E. coli strain targets glucosylated hydroxymethyl-C and may have evolved to cut T4 genome [78]. The resistance of hydroxymethyl-C-containing phage to restriction enzymes but its sensitivity to McrBC [79], the resistance of glucosylated hydroxymethyl-C to McrBC but sensitivity to GmrSD [78], and inhibition of GmrSD by T4-coded internal protein [78] suggest an evolutionary arms race (evolutionary struggle between competing sets of co-evolving genes that develop adaptations and counter-adaptations against each other) between the bacteria and the phage.

References

  • Fukuda E, Kaminska KH, Bujnicki JM, Kobayashi I. Cell death upon epigenetic genome methylation: a novel function of methyl-specific deoxyribonucleases. Genome Biology. 2008;9:R163. doi: 10.1186/gb-2008-9-11-r163. [PMC free article] [PubMed] [Cross Ref]

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