Unilateral PCA is common with much known about its associated signs and symptoms.1
In a study by Yamamoto et al
, aetiological mechanisms leading to PCA infarction included cardiac embolism, cryptogenic embolism, intrinsic PCA disease, vasoconstriction and coagulopathy.2
The aetiology of PCA territory infarction is unknown in more than a quarter of patients3
and no cause was identified in our case. The PCA supplies parts of the midbrain, the hippocampus, the thalamus, the mesial inferior temporal lobe, the occipital and the occipitoparietal cortices. It is also a source of collateral supply for the middle cerebral artery territory. Therefore features of PCA infarction may include visual, memory, sensory, psychological and motor deficits which may be transient or persistent in nature.3
Neau and Bogousslavsky have identified a syndrome of posterior choroidal artery territory infarction (PChA). The PChA originates from the second segment of the PCA.4
There are similarities between our case and Neau and Bogousslavsky's study in which one of 10 patients studied experienced short-term memory dysfunction and five of those 10 patients with PChA infarction had visual field deficits.5
Our case shows similarities to this syndrome.
- Posterior cerebral artery infarction may present with memory and visual field disturbances.
- These disturbances may be persistent in nature.
- The aetiology remains unknown in more than a quarter of cases.