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Cardiovasc Res. 2009 November 1; 84(2): 336.
PMCID: PMC2912656

Corrigendum to: Chlamydia heat shock protein 60 decreases expression of endothelial nitric oxide synthase in human and porcine coronary artery endothelial cells

[Cardiovascular Research, Volume 83, Issue 4, 1 September 2009, Pages 768–777; doi:10.1093/cvr/cvp150]

The authors of this study would like to acknowledge the contributions of F.F. Mussa to some of the data presented in Figure 6, which were in part previously published in an abstract,1 and to A.B. Lumsden for his critical discussions.

Reference

1. Mussa FF, Chai H, Lin PH, Lumsden AB, Yao QC, Chen CJ. Chlamydia pneumoniae heat shock protein-60 causes endothelial dysfunction through oxidative stress in porcine coronary arteries. J Surgical Res. 2006;130:202. (Abstract).

Articles from Cardiovascular Research are provided here courtesy of Oxford University Press